The following hormonal imbalances lead to overstimulation of the estrogen receptor and thus increase one’s risk for breast cancer :

1. Stress elevates cortisol, depletes dhea and results in a change in the immune system from a TH1 (normal bacterial and viral and cancer cell killing immune system) to a TH2 immune response (autoimmune type of response, and increased cancer risk type of immune response. Aging per se also changes the immune reponse to a Th2 response
2. Underactive thyroid and elevated insulin like in type 2 diabetes or the metabolic syndrome decreases SHBG and this increases the amount of free estrogen and causes overstimulation of the estrogen receptor
3. Xenoestrogens (ex ddt, exhaust fumes, estrogen farmfed chickens, plastics) we are all exposed to, and these increase our risk as well if we do not metabolize them properly and detoxify through the liver
4. Low progesterone. Progesterone deficiency occurs before estrogen deficiency in the perimenopause, and the risk for breast cancer goes up as the progesterone level goes down. Progesterone has antiproliferative effects on the breast and uterus. In antiaging medicine we never replenish estrogen without replenishing progesterone. Progesterone is protective against breast cancer, unlike progestins which potentially increase your risk for breast cancer. It is also recommended by antiaging physician’s to supplement progesterone even in hysterectomized women
5. Low melatonin ; high prolactin
6. Testosterone replenishment has been shown in trials to lower risk for breast cancer.

However too much testosterone like in polycystic ovarial syndrome can increase one’s estrogen level due to aromatization of the testosterone into estradiol with overstimulation of the estrogen receptor.
So to prevent breast cancer a good start would be to balance one’s hormones, lose weight if this is an issue and try and ensure that you metabolize estrogen in a healthy fashion

So let us take a closer look at the metabolism of estrogen in the liver.

Estrogen is metabolized through two pathways in the liver. The first phase detoxification in the liver is hydroxylation of the estrogen molecule, either to 2,4 or 16 hydroxyestrone. These molecules are fat soluble and in order to eliminate them you need to make these molecules water soluble, through phase 2 detoxification which is either through methylation, sulfation or glucoronidation.

2OH estrone is harmless, whereas 4OH and 16OH estrone are carcinogenic.

To increase the phase 1 production of 2OH estrone rather than 4 or 16OH estrone is a nice way to lower one’s risk for dangerous metabolites:

1. Cruciferous vegetables, I3C (Indole-3-carbinol, D-Indolemethane/DIM)
2. Moderate exercise
3. Whey protein
4. Soy
5. Omega 3 fish oil
6. Flax – whole and ground, not flax oil
7. Vit B6, B12, folic acid (in fact these simple vitamins protect form the dangers of alcohol use in increasing one’s risk for breast cancer)

However, taking I3C on its own is not sufficient since phase two detoxification needs to be supported as well. This phase two elimination is essential to excrete toxins and estrogen metabolites as well as for drug metabolism.
Methylation can be enhanced by methyl donors for example vit B6, vit B12, SAM-e, TMG.

The following nutrients increase glutathione:

1. NAC (N-acetylcysteine)
2. L-glutamine, glycine, mg, methionine, SAMe
3. Minimize depletion of glutathione with alpha lipoic acid, silymarin (milk thistle), gincko, whey protein
4. Brassica based diets (diets high in cruciferous vegetables)
5. Allium diet (leak/onion family – these increase GST (glutathione sulphur transferase function)

The following tests will be particularly helpful in our fight to prevent breast cancer:

1. Estrogen quotient (E3/(E2+E1) >1

2:16OH estrone ratio in the urine >2, <9 . More than 9 would increase one’s risk for osteoporosis since 16OH estrone is a potent estrogen whereas 2Oh estrone is a weak estrogen at the receptor site. 2OH estrone is also an antioxidant and a blood vessel protector

The new i-Breast Prevention combo by Integrative Intelligence Doctor Range that helps prevents the development of cancer in the breast tissue. This unique proprietary formula includes:

• Indole-3-carbinol from Broccoli
• CoQ10
• Calcium d-glucarate
• EGCG from green tea
• Selenium
• Vitamin D3
• Omega-s from fish oil
• certain other nutraceuticals

A recent study in humans indicated that vitamin E may significantly delay symptom onset and slow disease progression in MND because of its antioxidant properties. ⁵ Vitamin E helps protect cell membranes against lipid peroxidation.⁶ Selenium supplementation may increase the amount of vitamin E in the blood and increase activity of glutathione among MND patients. Ginkgo biloba also has antioxidant properties ⁷ and may promote healthy mitochondrial function. ⁸ During an in vitro study, Gingko biloba was found to protect against glutamate-induced excitotoxicity. ⁹

Zinc is involved in many physiological processes in the body. Changes in zinc metabolism that lead to neurodegeneration can occur during times of oxidative stress.¹⁰ Zinc supplementation has potential benefit due to its integral role in the function of SOD. SOD is an antioxidant enzyme that reduces oxidative stress and can play a key role in assisting MND. Mutations in SOD have decreased ability to bind to zinc and may contribute to MND. ¹¹ It is important to note that large doses of zinc inhibit copper absorption so therefore the dosage in supplementing zinc and the synergistic interaction of copper and zinc needs to be considered in terms of predicting the toxic or neuroprotective effects. ¹²

There is promise in using co-enzyme Q10 for treating MND. Co-enzyme Q10 is a critical component of the electron transport chain for proper mitochondrial function and acts as an antioxidant.¹³ A study found that oral supplementation significantly reduces weight loss, delays motor deficits, and extends life span in MND.¹⁴

Acetyl-l-Carnitine has been shown to inhibit mitochondrial damage and apoptosis. ¹⁵ It could also have neurotrophic activity and increase glutathione concentrations. ¹⁶ Early oral administration of acetyl-l-carnitine may significantly delay symptom onset, prolong motor function and extend survival rates. The effects of acetyl-l-carnitine are increased when administered in conjunction with alpha-lipoic acid.¹⁷

Alpha-lipoic acid, also an antioxidant may increase intracellular levels of glutathione ¹⁸, and chelate metals such as iron and copper.¹⁹ Furthermore, alpha-lipoic acid has been shown to protect cells against glutamate-induced excitotoxicity. ²⁰ Alpha-lipoic acid may therefore significant delay impaired motor performance as well as increase survival. ²¹

Dietary supplementation with amino acids may have some beneficial effects on the course of the disease.²² The use of creatine has also been encouraging. Creatine aids in the formation of ATP, which is the primary source of cellular energy. In multiple studies, creatine was shown to provide protective mechanisms against neurodegenerative diseases by helping to stabilize mitochondrial membranes and mitochondrial energy-transfer complexes.²³ Creatine may also reduce oxidative stress, increase glutamate uptake and improve motor performance. ²⁴ In addition, a small preliminary study found that creatine supplementation helps reduce the loss of muscle strength in MND patients.²⁵ Ultra high doses of vitamin B12 may also improve or slow muscle wasting, which is common during the later stages of the disease. ²⁶ The observation that patients with MND may have elevated plasma homocysteine levels has encouraged the use of the B vitamins, particularly folic acid and vitamin B12.

More recent data has suggested that curcumin (an extract from turmeric) may help improve calcium status in muscle tissue and reduce inflammatory processes. ^{27, 28} While human studies are needed to confirm these results, preclinical evidence suggests that curcumin could be useful in MND. Vinpocetine and Resveratrol may also inhibit the flow of calcium into cells, which is associated with glutamate-induced cell toxicity.^{29, 37} This is similar to the mechanism of action of riluzole, the only FDA-approved drug used to treat MND.

Evidence suggests that MND associated nerve cell death is partly due to low levels of the antioxidant glutathione, which protects cells from toxins and free radicals. ³⁰ Lower glutathione peroxidase activity has been shown in plasma and cerebrospinal fluid of MND patients. ³¹ Glutathione peroxidase, catalase, and superoxide dismutase (SOD) are all antioxidants synthesized by the body that counteract reactive oxygen species (ROS) damage. Toxicity resulting from mutated SOD is implicated in the pathophysiology in familial MND. Increasing glutathione levels could therefore help prevent free radical damage to cells. ³² The glutathione precursor N-acetyl cysteine (NAC) boosts blood levels of glutathione and oral administration decreases motor neuron loss, improves muscle mass, and increases survival time and motor performance.  ³³

Epigallocatechin gallate (EGCG) is a major catechin found in green tea. It displays antioxidant, anti-inflammatory ³⁴ and mild metal chelating properties. ³⁵ EGCG also significantly delays symptom onset and prolongs life span. Pycnogenol also has antioxidant properties ³⁶, as well as protective effects against glutamate excitotoxicity. ⁹ Procyanidins, which can be found in grape seeds, cranberries, blueberries, almonds, and peanuts, demonstrate potent antioxidant properties. ³⁶ Pycnogenol is a common complementary therapy option among MND patients. ⁶

Dehydroepiandrosterone (DHEA) is a naturally occurring steroid hormone and in several in vitro studies, DHEA has been shown to protect against glutamate-induced toxicity ³⁸ although only a few studies have examined DHEA in MND patients.

The benefits of aggressive nutritional support in affecting the course of disease and survival are well documented. Nutritional supplements that prevent free radical damage, stabilize mitochondrial membranes, or stimulate electron transport chain complexes may be of tremendous value in MND patients.

NUTRITIONAL SUPPORT PROTOCOL

Note: Suggested protocol for supplemental support only

References:

1. Mano Y, Takayanagi T, et al. [Amyotrophic lateral sclerosis and mercury—preliminary report]. Rinsho Shinkeigaku . 1990;30(11):1275–1277.
2. Lacomblez L, Bensimon G, et al. A confirmatory dose-ranging study of riluzole in ALS. ALS/Riluzole Study Group-II. Neurology. 1996;47(6 Suppl 4):S242–S250.
3. Rosenfeld J, Ellis A. Nutrition and Dietary Supplements in Motor Neuron Disease.Physical Medicine and Rehabilitation Clinics of North America, Volume 19, Issue 3, August 2008, Pages 573-589
4. Apostolski S, Marinkovic Z, et al. Glutathione peroxidase in amyotrophic lateral sclerosis: the effects of selenium supplementation. J Environ Pathol Toxicol Oncol . 1998;17(3–4):325–329.
5. Ascherio AJ, Weisskopf MG, et al. Vitamin E intake and risk of amyotrophic lateral sclerosis. Ann Neurol . 2005;57(1):104–110.
6. Cameron A, Rosenfeld J. Nutritional issues and supplements in amyotrophic lateral sclerosis and other neurodegenerative disorders. Curr Opin Clin Nutr Metab Care. 2002;5(6):631–643.
7. Ernst E. The risk-benefit profile of commonly used herbal therapies: Ginkgo, St. John’s Wort, Ginseng, Echinacea, Saw Palmetto, and Kava. Ann Intern Med. 2002;136(1):42–53.
8. Fosslien E. Mitochondrial medicine—molecular pathology of defective oxidative phosphorylation. Ann Clin Lab Sci. 2001;31(1):25–67.
9. Kobayashi MS, Han D, Packer L. Antioxidants and herbal extracts protect HT-4 neuronal cells against glutamate-induced cytotoxicity. Free Radic Res. 2000;32(2):115–124.
10. Cuajungco MP, Lees GJ. Zinc metabolism in the brain: relevance to human neurodegenerative disorders. Neurobiol Dis. 1997;4(3–4):137–169.
11. Banci L, Felli IC, et al. Direct detection of hydrogen bonds in monomeric superoxide dismutase: biological implications. Biochemistry. 2002;41(9):2913–2920.
12. Ermilova IP, Ermilov VB, et al. Protection by dietary zinc in ALS mutant G93A SOD transgenic mice. Neurosci Lett . 2005;379(1):42–46.
13. Galpern WR, Cudkowicz ME. Coenzyme Q treatment of neurodegenerative diseases of aging. Mitochondrion. 2007;7 suppl:S146–S153.
14. Beal MF. Coenzyme Q10 as a possible treatment for neurodegenerative diseases. Free Radic Res . 2002;36(4):455–460.
15. Carta A, Calvani M, et al. Acetyl-L-carnitine and Alzheimer’s disease: pharmacological considerations beyond the cholinergic sphere. Ann N Y Acad Sci. 1993;695:324–326.
16. Thal LJ, Calvani M, et al. A 1-year controlled trial of acetyl-1 -carnitine in early-onset AD. Neurology. 2000;55(6):805–810.
17. Hagen TM, Liu J, et al. Feeding acetyl-L-carnitine and lipoic acid to old rats significantly improves metabolic function while decreasing oxidative stress. Proc Natl Acad Sci U S A. 2002;99(4):1870–1875.
18. Zhang WJ, Frei B. Alpha lipoic acid inhibits TNF-alpha-induced NF-kappaB activation and adhesion molecule expression in human aortic endothelial cells. Faseb J . 2001;15(13): 2423–2432.
19. Pioro EP. Antioxidant therapy in ALS. Amyotroph Lateral Scler Other Motor Neuron Disord. 2000;1(Suppl 4):5–12; discussion 13–15.
20. Muller U, Krieglstein J. Prolonged pretreatment with alpha-lipoic acid protects cultured neurons against hypoxic, glutamate-, or iron-induced injury. J Cereb Blood Flow Metab. 1995;15(4):624–630.
21. Andreassen OA, Dedeoglu A, Friedlich A, et al. Effects of an inhibitor of poly(ADP-ribose) polymerase, desmethylselegiline, trientine, and lipoic acid in transgenic ALS mice. Experimental Neurology. 2001;168(2):419–424.
22. Palma A, de Carvalho M, et al. Biochemical characterization of plasma in amyotrophic lateral sclerosis: amino acid and protein composition. Amyotoph Lateral Scler Other Motor Neuron Disord . 2005;6(2):104–110
23. Persky AM, Brazeau GA. Clinical pharmacology of the dietary supplement creatine monohydrate. Pharmacol Rev. 2001;53(2):161–176.
24. Tarnopolsky MA, Beal MF. Potential for creatine and other therapies targeting cellular energy dysfunction in neurological disorders. Ann Neurol. 2001;49(5):561–574.
25. Mazzini L, Balzarini C, et al. Effects of creatine supplementation on exercise performance and muscular strength in amyotrophic lateral sclerosis: preliminary results. J Neurol Sci. 2001;191(1–2):139–144.
26. Kaji R, Kodama M, et al. Effect of ultrahigh-dose methylcobalamin on compound muscle action potentials in amyotrophic lateral sclerosis: a double-blind controlled study. Muscle Nerve . 1998;21(12):1775–1778.
27. Logan-Smith MJ, Lockyer PJ, et al. Curcumin, a molecule that inhibits the Ca2+-ATPase of sarcoplasmic reticulum but increases the rate of accumulation of Ca2+. J Biol Chem. 2001;276(50):46905–46911.
28. Sumbilla C, Lewis D, et al. The slippage of the Ca2+ pump and its control by anions and curcumin in skeletal and cardiac sarcoplasmic reticulum. J Biol Chem. 2002;277(16):13900–13906.
29. Wu SN. Large-conductance Ca2+- activated K+ channels: physiological role and pharmacology. Curr Med Chem . 2003;10(8):649–661.
30. Chi L, Ke Y, et al. Depletion of reduced glutathione enhances motor neuron degeneration in vitro and in vivo. Neuroscience. 9 Feb 2007;1443):991-1003
31. Chio A, Cucatto A, Terreni AA, Schiffer D. Reduced glutathione in amyotrophic lateral sclerosis: an open, crossover, randomized trial. Italian Journal of Neurological Sciences. 1998;19(6):363–366.
32. Exner R, Wessner B, et al. Therapeutic potential of glutathione. Wien Klin Wochenschr. 2000;112(14):610–616.
33. Andreassen OA, Dedeoglu A, et al. N-acetyl-L-cysteine improves survival and preserves motor performance in an animal model of familial amyotrophic lateral sclerosis. Neuroreport. 2000;11(11):2491–2493.
34. Hong JT, Ryu SR, et al. Neuroprotective effect of green tea extract in experimental ischemia-reperfusion brain injury. Brain Res Bull. 2000;53(6):743–749.
35. Levites Y, Youdim MB , et al. Attenuation of 6-hydroxydopamine (6-OHDA)-induced nuclear factor-kappaB (NF-kappaB) activation and cell death by tea extracts in neuronal cultures. Biochem Pharmacol. 2002;63(1):21–29.
36. Packer L, Rimbach G, et al. Antioxidant activity and biologic properties of a procyanidin-rich extract from pine (Pinus maritima) bark, pycnogenol. Free Radic Biol Med. 1999;27(5–6):704–724.
37. Wu SN, Li HF, et al. Vinpocetine-enhanced stimulation of calcium-activated potassium currents in rat pituitary GH3 cells. Biochem Pharmacol . 2001;61(7):877–892.
38. Lapchak PA, Araujo DM. Preclinical development of neurosteroids as neuroprotective agents for the treatment of neurodegenerative diseases. Int Rev Neurobiol. 2001;46:379–397.

• Provide structural support for the outer walls or membranes of the body’s cells
• Involved in cell to cell communication
• Helps with substance in the cells to pass into the blood
• Decreases inflammation
• Lowers triglycerides
• Raises HDL, (good) cholesterol
• Decreased blood pressure
• Enhances insulin action
• Needed for normal development and function of your brain, eyes, inner ear, adrenal glands, and reproductive tract
• Reduces PMS symptoms
• Help convert the nutrients from foods into usable forms of energy

• Make it possible for nutrients to pass from the blood through the cell walls
• Used to manufacture red blood cells
• Dilates or constricts blood vessels in the stomach, intestines, uterus, and bronchial tree
• Makes blood less sticky
• Decreases arrhythmias (irregular heart rhythm)
• May decrease homocysteine levels
• Helps protect against oxidation and ischemic heart disease
• Needed to make prostaglandins I and III which decrease in¬flammation, increase immune function, and decrease menstrual cramps
• Important for mitochondrial function (energy producing parts of your cells)

1. Age-related memory de¬clines
2. Asthma
3. Bumps on back of arms
4. Decreased memory and mental abilities
5. Dry hair, dandruff
6. Fatty infiltration of the liver
7. Heart disease
8. Increased cholesterol
9. Mood swings
10. Reduced vision
11. Sterility in men
12. Tingling or numbness of your arms or legs (neuropa¬thy)

13. Arthritis
14. Brittle nails, graying with horizontal splitting
15. Cracking skin at finger tips
16. Depression
17. Dry, scaly skin/dermatitis
18. Hair Loss
19. Impaired immune response
20. Miscarriage
21. Psychological disturbances
22. Slow wound healing
23. Thirst, excess urination

They are linoleic acid (omega 6 fatty acid) and linolenic acid (omega 3 fatty acid). Neither of these groups of fatty acids can be made by your body and therefore must be eaten or taken as a supplement

Your body requires omega-6-fatty acids to maintain your health. However if you intake too many of them you cause in¬flammation by producing prostaglandins that go down an inflammatory pathway instead of an anti-inflammatory path¬way.  Our standard diet is very low in omega-3-fatty acids and very high in omega-6-fatty acids. It is best to intake three to six parts of omega-6 fatty acids to one part omega-3¬fatty acids instead of what most of us eat which is between 10:1 to 25:1.

Your body requires zinc, magnesium, niacin, vitamin C, vita¬min A, biotin, B vitamins, and other nutrients to convert fatty acids.

Nutritional research suggests that omega-7s hold promise for cardiovascular and brain health as well as better fat metabolism in general.

One of the best known omega-7s is palmitoleic acid, or its ester form palmitoleate. In its cis-isomer form, it is a monounsaturated fatty acid (MUFA).

Common food sources of omega 7 include :
Dietary Source Palmitoleic Acid (g/100 g of Fatty Acids)

• Macadamia nut oil 17.3%
• Cod liver oil 7.1%
• Salmon 6.0%
• Olive oil 1.4%
• Eggs 0.3%
• Soybean oil 0.08%

Only recently has palmitoleic acid been identified as having a major role in protection from cardiovascular disease.      Omega 7 also helps with cell membrane health in the form of phospholipid synthesis. Palmitoleic acid can be produced in the human body and then used to make triglycerides.

Numerous studies show that omega 7’s are good for our lipid profile and cardiovascular health :

SynerChi Organics – Omegas in Hemp Foods, by Kiara & Ethan Ducasse

Omegas are the essential fatty acids that our bodies need to function optimally, both mentally and physically throughout our stressful days. This is why we recommend using hemp foods as these have a perfectly balanced ratio of omegas 3, 6 & 9 in a 3:1:1 ratio. Omega 3 is a polyunsaturated fat and has incredible benefits which exceed those of any other fats. Some of the health benefits of Omegas are:

• Protects memory, brain and nervous system performance
• Reduces the risk of heart disease and stroke
• An anti-inflammatory that eases arthritis, joint pain and improves skin conditions
• Promotes a healthy pregnancy
• Reduces and fights the symptoms of depression and improves emotional health
• Promotes weight  loss

Omegas successfully reduce inflammation and rebuilds healthy brain and nervous system tissue. A high-quality plant-based omega source is something that comes highly recommended for our overall health of our bodily functions.

When your omega intake is inadequate, your nerve cells become stiff and more prone to inflammation. Once your nerve cells become rigid and inflamed proper neurotransmission (communication) between our cells become erratic and compromised. Omegas improve your mood and memory, giving you protection against brain disorders ranging from depression to dementia, and also assist the heart in staying healthy and clean from cholesterol. Omegas lower your risk and treat some major chronic illnesses including heart disease, cancer, and arthritis.

It was found that a quality nutritional omega oil blend with the correct 2:1:1 ratio of omega-3 and omega-6 (EFAs) and omega-9 at the rate of 15ml per 25kg of bodyweight per day will:

• Enhance stamina by 40 – 60%
• Improves muscular development (anabolic)
• Quicker recovery from fatigue
• Speed healing of injuries
• Improves our concentration, skin conditions, cardiovascular functions, quality of sleep, and lowers insulin requirements in people with type 1 diabetes.

The formation of eicosanoids – molecules which exert control over bodily systems such as immunity and inflammation act as a molecular messenger in the central nervous system. Prostaglandins are of the most importance to muscle growth. They increase sensitivity to insulin, help to maintain normal levels of primary testosterone enhance the male androgen hormone, increases the body’s secretion of growth hormone, and an increase in the synthesis of protein to further build muscle cells.

Belly fat in particular is especially dangerous and is considered to be more predictive of poor health than body mass index (BMI). Low omega-3 fatty acids also play a role in the accumulation of belly fat. In fact, higher omega-3 fatty acid levels may reduce belly fat by decreasing fat cell size. These results suggest that dietary management with omega-3 fatty acids may represent an important way to better manage BMI and belly fat.

Adding omega fatty acids to our lifestyle can improve the body’s composition by combining plant based nutrients with a calorie restricted, active lifestyle to accelerate our weight loss process. This is where hemp comes in as it is low in carbohydrates and has a seemingly suppressive “placebo” effect on our appetites by providing our bodies with the correct nutrients which leads to the consumption of fewer calories, fat and sugars.

• Omega 3 fatty acids EPA and DHA lower your insulin levels and any extra calories you eat are burned off for energy, instead of being stored for later use (as body fat)
• All omega 3 sources are anti-lipogenic (prevents blockages of fat storage in your body) and increase your metabolic rate, so you burn body fat faster
• Omega 3 oils lower the cortisol levels, a stress-related hormone that causes you to store rather than burn off extra calories
• Omega 3 fatty acids are proven to improve the blood flow to muscles during exercising, thus increasing your body’s ability to burn more fat
• Eating foods rich in omega 3 or using them as supplements normalizes our blood sugar levels and as a result, you have more energy and drive to exercise

Omega three fatty acids improve your liver’s ability to burn fat, process toxins and keep the blood system clean and healthy

Dietary fats impact gut bacteria, omega-3 polyunsaturated fatty acids (PUFAs) and omega-6 (PUFAs). The omega-3 EPA and DHA found in hemp seed oil, hemp hearts and other proteins reduces inflammation in the gastric system and increase the benefit of microorganisms to protect against gastrointestinal (GI) diseases. These findings were presented at the 11^th Congress of the International Society for the Study of Fatty Acids and Lipids (ISSFAL) in Stockholm 1 July, 2014.
Dietary choices and certain microorganisms in the GI tract can contribute to the prevention or development of inflammatory bowel disease, colitis (inflammation of the colon) and Crohn’s disease. PUFAs in particular affect microbes living in the intestine known as “gut microbiota.”

• Assists in digestive system function
• Lowers blood pressure & cholesterol
• Increases metabolic rate
• Reduction in cravings of unhealthy foods
• Naturally containing CLA & GLA
• Enhances our GI health

More frequently we are getting positive feedback about the quality hemp foods bring to our daily lifestyles amplifying, protecting and strengthening our immune system and bodies.
Hemp foods are one of the worlds’ most concentrated and balanced sources of proteins, enzymes, essential fatty acids and vitamins found in our food source.
The broad function of Hemp Hearts is of unsurpassed promotion of longevity and health within the human body. This fascinating seed promotes vigorous cellular development, lowering the risk of intestinal diseases, likely reducing the risk of colon cancer.

Many factors come into play in this tricky question and age, weight, lifestyle habits and your health. According to research done by the USDA an individual should:

• Avoid trans-fats, or keep to only 1 % of your 2000 calories or 2 grams. These fats become deformed during a process called hydrogenation, making the oil more stable reducing any spoil. Good for manufacturers, certainly not good for you. This may lead to many heart problems and possibly cancer.

One very important aspect to understand about these fats is that good fats can become bad fats if they are exposed to high temperatures, light, oxygen which will damage these fats. These types of fats should be refrigerated to protect the nutritional values. When using these fats in cooking, be sure not to overheat the oils as some of them may become carcinogenic.

In ending, we find that we should focus on incorporating the right omegas into our diets, eliminating trans fats and minimising saturated fats. Omegas are highly important for the optimal function of your mental and physical performance, assisting in faster recovery, better sleep and quality of life.

May this information inspire you to take better care of your temple, further expand your knowledge, and share valuable information with your loved ones.

Please be in touch with the SynerChi Organics team to find out more information on what we will be getting up too from here www.synerchiorganics.co.za